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J Nephropathol. Inpress.
doi: 10.34172/jnp.2022.17241
  Abstract View: 1238

Original Article

Histological changes associated with early and late renal allograft dysfunction in a large three-center transplant program in Iraq

Alaa Abbas Ali 1* ORCID logo, Dana A Sharif 2 ORCID logo, Safaa E Almukhtar 3

1 Department of Pathology, University of Sulaimani College of Medicine and Shoresh Teaching Hospital, Sulaimani, Iraq
2 Department of Nephrology, University of Sulaimani College of Medicine, Sulaimani, Iraq
3 Department of Nephrology, University of Hawler College of Medicine, Erbil, Iraq
*Corresponding Author: *Corresponding author: Alaa Abbas Ali, Email: , Email: dr.alaaabbas@yahoo.com

Abstract

Introduction: Transplantation is the sole viable option for the long-term survival of patients with end-stage renal disease (ESRD) in low-resourced countries.

Objectives: To report the histopathological characteristics of kidney graft dysfunction in a large transplant program of a developing country.

Patients and Methods: Renal transplant biopsies were analyzed by the Banff 2017 classification and subdivided into early (≤1 year) or late (>1 year) post-engraftment periods during the 12 months of 2019.

Results: Here, 290 satisfactory graft biopsies were obtained on 290 patients for graft failure and/ or proteinuria. The median age of the recipient was 39 years (interquartile range 28-47), where 77% were male and 5.5% had been previously transplanted and 84% of donors were unrelated. Histological diagnosis was as follow; acute T-cell mediated rejection (A-TCMR; 23.1%), acute tubular necrosis (ATN; 14.8%), interstitial fibrosis and tubular atrophy (IFTA; 11.4%), recurrent or de novo kidney disease (R/DKD; 8.6%), transplant glomerulopathy (TG; 7.6%), calcineurin inhibitor toxicity (CNI; 6.9%), and active antibody-mediated rejection (A-AMR; 8.6%). Early graft dysfunctions were A-TCMR (29%) and ATN (22.4%). Late graft dysfunction included IF/TA, (20.2%), TG (20.2%), R/DRD (17%), and A-TCMR (9.5%). C4d+AMR was equally represented in early (5.6%) and late (6.3%) biopsies.

Conclusion: A-TCMR was the most common cause of early graft dysfunction and was replaced by chronic conditions as the cause of 57.8% of late graft biopsies. The causes of graft dysfunction are not remarkably different from the west and TG will be a major cause of late graft failure in Iraq.


Implication for health policy/practice/research/medical education:

In our study on 290 renal allograft recipients, acute cellular rejection was the most common cause of graft dysfunction and peaked in the first year post-transplantation while interstitial fibrosis and tubular atrophy (non-otherwise specified) and transplant glomerulopathy occurred mainly after the first year of transplantation.

Please cite this paper as: Ali AA, Sharif DA, Almukhtar SE. Histological changes associated with early and late renal allograft dysfunction in a large three-center transplant program in Iraq. J Nephropathol. 2022;11(x):exx. DOI: 10.34172/jnp.2022.xx.

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Submitted: 07 Jun 2021
Accepted: 09 Feb 2022
ePublished: 18 Feb 2022
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