﻿<?xml version="1.0" encoding="UTF-8"?>
<ArticleSet>
  <Article>
    <Journal>
      <PublisherName>Society of Diabetic Nephropathy Prevention</PublisherName>
      <JournalTitle>Journal of Nephropathology</JournalTitle>
      <Issn>2251-8363</Issn>
      <Volume>8</Volume>
      <Issue>1</Issue>
      <PubDate PubStatus="ppublish">
        <Year>2019</Year>
        <Month>01</Month>
        <DAY>01</DAY>
      </PubDate>
    </Journal>
    <ArticleTitle>Renoprotective impact of angiotensin 1-7: Is it certain?</ArticleTitle>
    <FirstPage>e01</FirstPage>
    <LastPage>e01</LastPage>
    <ELocationID EIdType="doi">10.15171/jnp.2019.01</ELocationID>
    <Language>EN</Language>
    <AuthorList>
      <Author>
        <FirstName>Mehdi</FirstName>
        <LastName>Nematbakhsh</LastName>
      </Author>
    </AuthorList>
    <PublicationType>Journal Article</PublicationType>
    <ArticleIdList>
      <ArticleId IdType="doi">10.15171/jnp.2019.01</ArticleId>
    </ArticleIdList>
    <History>
      <PubDate PubStatus="received">
        <Year>2018</Year>
        <Month>02</Month>
        <Day>07</Day>
      </PubDate>
      <PubDate PubStatus="accepted">
        <Year>2018</Year>
        <Month>06</Month>
        <Day>29</Day>
      </PubDate>
    </History>
    <Abstract>The two important arms of renin angiotensin system (RAS) are angiotensin II (Ang II) and angiotensin1-7 (Ang1-7). Both of these peptides are present in the kidney, while the renal hemodynamic responses to these peptides act differently in kidney circulation. For this short-review, we used a variety of sources including PubMed, Google Scholar, and Scopus. Although in normal physiological condition, Ang1-7 has been known as an inactive agent in the renal system, however in past years many experimental and clinical reports indicated the protective role of Ang1-7 in renal hemodynamics and functions under different circumstances. In the current article, the possible renoprotective role of Ang1-7 was briefly reviewed.</Abstract>
    <ObjectList>
      <Object Type="keyword">
        <Param Name="value">Renin angiotensin system</Param>
      </Object>
      <Object Type="keyword">
        <Param Name="value">Angiotensin II</Param>
      </Object>
      <Object Type="keyword">
        <Param Name="value">Angiotensin1-7</Param>
      </Object>
      <Object Type="keyword">
        <Param Name="value">Vascular endothelium</Param>
      </Object>
      <Object Type="keyword">
        <Param Name="value">Kidney injury</Param>
      </Object>
      <Object Type="keyword">
        <Param Name="value">Bradykinin receptors</Param>
      </Object>
    </ObjectList>
  </Article>
</ArticleSet>